Saturday, July 11, 2009

Fever


Children having fever are cause of stress, worries and sleeplessness among parents, If you are one one them you know what I really mean.

If a child has a fever most of us immediately give Tylenol, Paracetamol, Aspirin or other over-the counter medications. Of course we would rather choose chewable tablets or flavored syrup especially designed for children. Is it a right thing to do?

This article provides educational information about the role of fever in our bodies and how it can be safely controlled. This information is not a substitute for your doctor’s advice.

What Humans have an internal system controlled by the hypothalamus, which maintains our body temperature around 98.6 degrees F. The “normal,” range fluctuates from 98.6 degrees to 99.5 degrees F, which is an optimal range for all biochemical reactions in the body. A fever is when the body’s temperature rises above 100.5 degrees F.
Although in most cases fever develops in response to infections, cancer, and disease; it can also appear after a vigorous exercise. Fever itself is not a disease but a symptom of an underlying pathological process and in many cases does not require medical intervention unless it is higher than 102 degrees F. The increased body temperature helps in fighting the infection; therefore a child with a fever who is feeling and behaving well may not need any medication, unless he/she is younger than 3 months.

Increase in our body temperature is a defense mechanism by which the body cells shut down many functions and shift their activity towards producing a series of specific “safety” proteins called heat shock proteins. Although scientists are still studying their many functions, it is known that heat shock proteins are critical for survival when any organism is exposed to stress. These proteins protect us not only against damage by heat, but also by toxins, heavy metals, oxidative agents or a lack of oxygen (hypoxia). Therefore, a temporary increase in the body’s temperature causes little harm and it is needed to launch this natural defense.

Consequently, rushing with the medication at the first sign of fever is not always beneficial and even tends to prolong the illness rather than shortening it. Studies show that moderate fever can aid in the fight against infectious organisms and boosting the body’s immune response. Parents should not worry excessively about fever in children who are eating, drinking, and acting normally. If the fever rises to 103 degrees F, or the child becomes uncomfortable, consultation with a physician is necessary.

Info above was taken from Dr. Rath Research Institute.

Sunday, July 5, 2009

Walnut


Walnuts 'combat unhealthy fats'

The new superfood?
Eating walnuts at the end of a meal may help cut the damage that fatty food can do to the arteries, research suggests.
It is thought that the nuts are rich in compounds that reduce hardening of the arteries, and keep them flexible.
A team from Barcelona's Hospital Clinico recommend eating around eight walnuts a day.
The study, which appears in the Journal of the American College of Cardiology, also showed walnuts had more health benefits than olive oil.
The protective fat from walnuts actually undoes some of the detrimental effects of a high-saturated- fat diet

Professor Robert Vogel
The researchers recruited 24 adults, half with normal cholesterol levels, and half with levels that were moderately high to the research, which was partly funded by the California Walnut Commission.
Each was given two high-fat salami and cheese meals, eaten one week apart.
For one meal, the researchers added five teaspoons of olive oil. For the other, they added eight shelled walnuts.
Tests showed that both the olive oil and the walnuts helped to reduce the sudden onset of harmful inflammation and oxidation in arteries that follows a meal high in saturated fat.
Over time, this is thought to cause the arteries to start to harden - and increase the risk of heart disease and stroke.
However, unlike olive oil, adding walnuts also helped preserve the elasticity and flexibility of the arteries, regardless of cholesterol level.
Arteries that are elastic can expand when needed to increase blood flow.
Lead researcher Dr Emilio Ros said eating high fat meals disrupted production of nitric oxide by the inner lining of the arteries, a chemical needed to keep blood vessels flexible.
Key chemical
Walnuts contain arginine, an amino acid used by the body to produce nitric oxide.
The nuts also contain antioxidants and alpha-linolenic acid (ALA), a plant-based omega-3 fatty acid with health giving properties.
Dr Ros is starting a new trial to see whether the ALA in walnuts can help people with abnormal heart rhythms.
He warned against people assuming they can eat what they like so long as they accompany it with walnuts.
"Instead, they should consider making walnuts part of a healthy diet that limits saturated fats."
Professor Robert Vogel, of University of Maryland in Baltimore, said: "This demonstrates that the protective fat from walnuts actually undoes some of the detrimental effects of a high-saturated- fat diet, whereas a neutral fat, such as olive oil, does not have as much protective ability.
"This raises a very interesting issue because many people who eat a Mediterranean diet believe the olive oil is providing the benefits.
"But this research and other data indicate that's not true.
"There are probably other factors in the diet, including that it is a relatively rich source of nuts.
"This is not to say that olive oil is bad, but it's not the key protective factor in the Mediterranean diet."

Taken From: BBC news channel, posted by Dr. Allan Cadano

Monday, June 29, 2009

Low Dose Naltexone and Cancer Part 4




Among those who have shown significant movement toward remission, most had never received chemotherapy. The apparent remissions:
2 children with neuroblastoma
6 patients with non-Hodgkin's lymphoma
3 with Hodgkin's disease
5 with pancreatic cancer metastatic to the liver
5 with multiple myeloma
1 with carcinoid
4 with breast cancer metastatic to bone
4 with ovarian cancer
18 with non-small cell cancer of the lung
1 with small cell cancer of the lung
5 with prostate cancer (no prior hormone-blocking therapy)

(Although recently-diagnosed prostate cancer patients who have not received other therapies appear to do well on LDN, patients with prostate cancer who have already been treated with hormone-related therapies, including testosterone-blocking drugs and PC-Spes, have not responded to LDN.)
An overview of these results must assume the basic statistical principle that the patients with no follow-up contact have not done as well as those who have maintained continual medical contact with Dr. Bihari. Measured in terms of disease stabilization and/or movement toward remission, and assuming that patients in continual follow-up are twice as likely to have had a good outcome thus far, it appears that over one-half of all cancer patients whom Dr. Bihari has started on LDN have done well.
Taking into account the relatively large number of patients who were in advanced stages of disease when first seen by Dr. Bihari, and that some patients in the "not followed up" and "LDN < 6 mos" groups will likely have positive outcomes, it appears possible that more than 60% of patients with cancer may significantly benefit from LDN. This is underscored by Dr. Bihari's observation that better outcomes tend to be seen when treatment with LDN is begun in earlier stages of the disease. Of interest, there is a negligible rate of relapse in patients who are started on LDN after or during successful initial treatment with surgery (e.g., for breast cancer) or with chemotherapy (e.g., for Hodgkin's disease or non-Hodgkin's lymphoma).
It will clearly require extensive study of LDN in prospective, controlled clinical trials to determine which cancers respond best and which other therapies are complementary to or synergistic

Source:http://www.lowdosenaltrexone.org

Monday, June 22, 2009

Low Dose Naltexone and Cancer Part 3


Recent Developments
> As of March 2004
Since February 1999, Dr. Bihari has begun treatment of some 450 cancer patients with LDN. Since many of these patients, particularly those seen before October 2000, were seen only once in consultation with medical follow-up by their oncologists, Dr. Bihari is missing up-to-date follow-up data on 96 patients.
As of March 2004, of the remaining 354 patients, 84 have died, all but 4 of cancer-related causes. Most of these deaths have occurred in the first 8 to 12 weeks on LDN. For the most part, these were patients who were quite ill when first seen, and had exhausted all other treatment possibilities. Of the remaining 270 patients, 220 have been on LDN for six months or longer. Of these, 86 have shown significant movement toward remission, identified for this purpose as a reduction of at least 75% in tumor mass and tumor-related symptoms. Of the other 134 patients, 9 have continued to show tumor progression, whereas the other 125 have stabilized and/or are moving toward remission but do not yet meet the 75% reduction criterion.

Monday, June 15, 2009

Low Dose Naltexone and Cancer Part 2


Cancers that are reported by Dr. Bihari to apparently respond to LDN:
Bladder Cancer
Breast Cancer
Carcinoid
Colon & Rectal Cancer
Glioblastoma
Liver Cancer
Lung Cancer (Non-Small Cell)
Lymphocytic Leukemia (chronic)
Lymphoma (Hodgkin's and Non-Hodgkin's) Malignant Melanoma
Multiple Myeloma
Neuroblastoma
Ovarian Cancer
Pancreatic Cancer
Prostate Cancer (untreated)
Renal Cell Carcinoma
Throat Cancer
Uterine Cancer > What the Future Holds
If the results of trials of low dose naltrexone in certain cancers are positive, the drug could eventually become an additional mainstay of cancer treatment ? adjunctive with chemotherapy, radiation, and other cancer cell growth inhibitor receptor agonists ? or even a replacement for current therapies, as primary treatment for those cancers that show little response to standard therapies.

Monday, June 8, 2009

Low Dose Naltexone and Cancer Part 1


LDN and Cancer
In Brief  Recent Developments  Noteworthy Cases  Background  LDN Homepage

In Brief
Although prospective, controlled clinical trials on LDN in the treatment of cancer are yet to be accomplished, as of March 2004 clinical "off-label" use of this medication by Dr. Bihari in some 450 patients with cancer almost all of whom had failed to respond to standard treatments suggests that more than 60% of patients with cancer may significantly benefit from LDN.
Of the 354 patients with whom Dr. Bihari had regular follow-up, 86 have shown objective signs of significant tumor shrinkage, at least a 75% reduction. 125 patients have stabilized and/or are moving toward remission.
Dr. Bihari's results sharply contrast to prior usual cancer treatment outcomes: either a cancer-induced death or a total cure. LDN therapy presents a viable third alternative, the possible long-term stabilization and/or gradual reduction of tumor mass volume.
Thus, with LDN, cancer can — in some cases — become a manageable chronic disease. Patients have the possibility of living free of symptoms, without, in many cases, the crippling side-effects of chemotherapy and radiation treatment.
> How It Works
Low dose naltrexone might exert its effects on tumor growth through a mix of three possible mechanisms:
1.By inducing increases of metenkephalin (an endorphin produced in large amounts in the adrenal medulla) and beta endorphin in the blood stream;
2.By inducing an increase in the number and density of opiate receptors on the tumor cell membranes, thereby making them more responsive to the growth-inhibiting effects of the already-present levels of endorphins, which induce apoptosis (cell death) in the cancer cells; and
3.By increasing the natural killer (NK) cell numbers and NK cell activity and lymphocyte activated CD8 numbers, which are quite responsive to increased levels of endorphins.

Monday, June 1, 2009

Ellagic Acid Part3

Ellagic Acid Study - The Hollings Cancer Institute at the University of South Carolina has conducted a double blind study on a group of 500 cervical cancer patients that had everyone excited. Nine years of study have shown that a natural product called Ellagic acid is causing G-arrest within 48 hours (inhibiting and stopping mitosis-cancer cell division), and apoptosis (normal cell death) within 72 hours, for breast, pancreas, esophageal, skin, colon and prostate cancer cells.
Ellagic Acid Clinical Tests on cultured human cells also show that Ellagic acid prevents the destruction of the p53 gene by cancer cells. Additional studies suggest that one of the mechanisms by which Ellagic acid inhibits mutagenesis and carcinogenesis is by forming adducts with DNA, thus masking binding sites to be occupied by the mutagen or carcinogen. Ellagic acid can be found in different foods. The Hollings Clinic has identified the red raspberry for their studies."

Ellagic Acid is astounding cancer researchers worldwide with it's ability to apparently prevent many types of cancer cells from multiplying, thus causing the cancer to die. This compound is found in berries.
Experience the synergistic effects of five super fruits (mangosteen, acai, pomegranate, noni, goji) plus resveratrol. Loaded with antioxidants that support higher energy levels and a stronger immune system. Also includes proprietary fruit blend from raspberries, wild cherry, blueberry, elderberry and grapeseed extract.

Source: http://alternativecancer.us